Friday, November 30, 2012

Self Inspiration Now | The Secret & the Law of Attraction


Self Help ? It Is All About You

Everyone is full of competition which is also moving forward in every important categories. Additionally it is important for everyone to stay different among other to reach your goals in business and in your life. In order to meet this issue you should have self improvements actions inside your every attitude. Self improvements assist one person inside the personality development. Additionally it is important for those who wish to be unique in everyone. Michael Humphries It?s All Good.

Personal development coaches help the people to fulfill the area through which they may be lacking and required improvement. The coaches will guide with all kinds of instructions and direction to turn into a well man. You can find kinds of personal development techniques are accompanied by many people being successful. First step involved on taking adequate rest. A relaxed free mind will assist to obtain better ideas. A good rest will assist you to think well even at the time busy schedule. Additionally it is helpful to make fresh objectives and goals for yourself to realize. In the event you continue in one condition or status, other will advance ahead you in from the personal and business life.

Self improvements also included with setting new objectives and goals with correct planning.. You have to provide little focus on your thoughts. You need to consider all the stuff before implementation. Making all kinds of analysis and considering all kinds of possibilities in all of the works will assist you to improve as well as the same will displayed inside your performance. It?s not necassary to disturb or become sad along with your past failures. You have to learn from those mistakes along with to behave to reduce this error in further actions. Failures help learn specific things in fact it is the principle step which makes to obtain success. Additionally it is good to make a good relationship online websites. It must be both in the office as well as life. A fantastic communication will remove all sorts of jealousy, greediness and develop a soft kind mind. While performing and dealing success for your life, you must spend time with your friends also. It will help to chill and also to take advantage of the old memories which will allow you to refresh. You have to have a good balance between business as well as life. You can also develop oneself improvement by reading more books from your popular writers. No Deposit Electricity

The way to Develop positive Thoughts:

People thoughts are so powerful and the thoughts also. Greater people think about one, greater people are attracted to do on the action. So we will need to take care with the thoughts and it is also good to reduce thoughts that are negative. Having positive thoughts brings excellent results in the life. It is going to give comfort and let you becoming a likeable person towards others. it helps to build good relationships and keeping an optimistic energy will lead an excellent life. Learning how to stay positive within the way of life can be impossible at specific times particularly when frustrations and troubles are looming. However when you develop a positive thought slowly, you can easily conquer those ideas and carry command the life span and grow positive. Study affirmations, think positive, have confidence in yourself, suppose the things in terms you should live help others and execute action to attain your goals. You can also begin from small you to definitely the manageable tasks. It is usually good to be with the attitudes always. If any troubles will come in the right path, grow from it how to handle it and what should not do etc. it will help you to lead an excellent life, merely a self improvement will give good results. It?s All Good, Michael Humphries Book Check this out ! Cheap Dental Plan

Source: http://www.drbonomi.com/enblog/self-inspiration-now/

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ScienceDaily: Gene News

ScienceDaily: Gene Newshttp://www.sciencedaily.com/news/health_medicine/genes/ Genes and Genetics News. Read today's medical research in genetics including what can damage genes, what can protect them, and more.en-usWed, 28 Nov 2012 04:58:13 ESTWed, 28 Nov 2012 04:58:13 EST60ScienceDaily: Gene Newshttp://www.sciencedaily.com/images/logosmall.gifhttp://www.sciencedaily.com/news/health_medicine/genes/ For more science articles, visit ScienceDaily.How infidelity helps nieces and nephews: Men may share more genes with sisters' kids than cheating wife's kidshttp://www.sciencedaily.com/releases/2012/11/121127190021.htm A new study produced new mathematical support for a theory that explains why men in some cultures often feed and care for their sisters? children: where extramarital sex is common and accepted, a man?s genes are more likely to be passed on by their sister?s kids than by their wife?s kids.Tue, 27 Nov 2012 19:00:00 ESThttp://www.sciencedaily.com/releases/2012/11/121127190021.htmGene linked to respiratory distress in babieshttp://www.sciencedaily.com/releases/2012/11/121127130256.htm Some infants are more susceptible to potentially life-threatening breathing problems after birth, and rare, inherited DNA differences may explain why, according to new research.Tue, 27 Nov 2012 13:02:02 ESThttp://www.sciencedaily.com/releases/2012/11/121127130256.htmNew understanding of X chromosome inactivationhttp://www.sciencedaily.com/releases/2012/11/121127101534.htm Scientists have broadened our understanding of how cells regulate silencing of the X chromosome in a process known as X-inactivation.Tue, 27 Nov 2012 10:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121127101534.htmNew mechanism for cancer progression discoveredhttp://www.sciencedaily.com/releases/2012/11/121127094311.htm Researchers have discovered an alternative mechanism for activating rhe oncogene Ras that does not require mutation or hormonal stimulus.Tue, 27 Nov 2012 09:43:43 ESThttp://www.sciencedaily.com/releases/2012/11/121127094311.htmProtein injection points to muscular dystrophy treatmenthttp://www.sciencedaily.com/releases/2012/11/121127094248.htm Scientists have discovered that injecting a novel human protein into muscle affected by Duchenne muscular dystrophy significantly increases its size and strength, findings that could lead to a therapy akin to the use of insulin by diabetics.Tue, 27 Nov 2012 09:42:42 ESThttp://www.sciencedaily.com/releases/2012/11/121127094248.htmChemical 'switches' for neurodegenerative diseases discoveredhttp://www.sciencedaily.com/releases/2012/11/121127093951.htm Researchers have identified and ?switched off? a chemical chain that causes neurodegenerative diseases such as Huntington?s disease, amyotrophic lateral sclerosis and dementia. The findings could one day be of particular therapeutic benefit to Huntington?s disease patients.Tue, 27 Nov 2012 09:39:39 ESThttp://www.sciencedaily.com/releases/2012/11/121127093951.htmGene that causes tumor disorder linked to increased breast cancer riskhttp://www.sciencedaily.com/releases/2012/11/121127093855.htm New research showing a more than four-fold increase in the incidence of breast cancer in women with neurofibromatosis-1 (NF1) adds to growing evidence that women with this rare genetic disorder may benefit from early breast cancer screening with mammograms beginning at age 40, and manual breast exams as early as adolescence.Tue, 27 Nov 2012 09:38:38 ESThttp://www.sciencedaily.com/releases/2012/11/121127093855.htmMetabolic protein launches sugar feast that nurtures brain tumorshttp://www.sciencedaily.com/releases/2012/11/121126164003.htm PKM2 slips into nucleus to promote cancer; potential biomarker and drug approach discovered.Mon, 26 Nov 2012 16:40:40 ESThttp://www.sciencedaily.com/releases/2012/11/121126164003.htmPossible new treatment for Ewing sarcomahttp://www.sciencedaily.com/releases/2012/11/121126142855.htm Discovery of a new drug with high potential to treat Ewing sarcoma, an often deadly cancer of children and young adults, and the previously unknown mechanism behind it, come hand-in-hand in a new study.Mon, 26 Nov 2012 14:28:28 ESThttp://www.sciencedaily.com/releases/2012/11/121126142855.htmSurvival gene may be key to controlling HIV and hepatitishttp://www.sciencedaily.com/releases/2012/11/121126131349.htm A newly discovered gene that is essential for embryo survival could also hold the key to treating and potentially controlling chronic infections such as HIV, hepatitis and tuberculosis. The gene, called Arih2, is fundamental to the function of the immune system -- making critical decisions about whether to switch on the immune response to an infection.Mon, 26 Nov 2012 13:13:13 ESThttp://www.sciencedaily.com/releases/2012/11/121126131349.htmMicrobial 'missing link' discovered after man impales hand on tree branchhttp://www.sciencedaily.com/releases/2012/11/121126110737.htm Two years ago, a 71-year-old Indiana man impaled his hand on a branch after cutting down a dead tree. The wound caused an infection that led scientists to discover a new bacterium and solve a mystery about how bacteria came to live inside insects.Mon, 26 Nov 2012 11:07:07 ESThttp://www.sciencedaily.com/releases/2012/11/121126110737.htmTransposable elements reveal a stem cell specific class of long noncoding RNAshttp://www.sciencedaily.com/releases/2012/11/121125192838.htm Over a decade after sequencing the human genome, it has now become clear that the genome is not mostly ?junk? as previously thought. In fact, the ENCODE project consortium of dozens of labs and petabytes of data have determined that these ?noncoding? regions house everything from disease trait loci to important regulatory signals, all the way through to new types of RNA-based genes.Sun, 25 Nov 2012 19:28:28 ESThttp://www.sciencedaily.com/releases/2012/11/121125192838.htmNew molecular culprit linked to breast cancer progressionhttp://www.sciencedaily.com/releases/2012/11/121124090511.htm Researchers have uncovered a protein ?partner? commonly used by breast cancer cells to unlock genes needed for spreading the disease around the body. A report on the discovery details how some tumors get the tools they need to metastasize.Sat, 24 Nov 2012 09:05:05 ESThttp://www.sciencedaily.com/releases/2012/11/121124090511.htmNew insights into virus proteome: Unknown proteins of the herpesvirus discoveredhttp://www.sciencedaily.com/releases/2012/11/121123092132.htm The genome encodes the complete information needed by an organism, including that required for protein production. Viruses, which are up to a thousand times smaller than human cells, have considerably smaller genomes. Using a type of herpesvirus as a model system scientists have shown that the genome of this virus contains much more information than previously assumed. The researchers identified several hundred novel proteins, many of which were surprisingly small.Fri, 23 Nov 2012 09:21:21 ESThttp://www.sciencedaily.com/releases/2012/11/121123092132.htmScientists describe elusive replication machinery of flu viruseshttp://www.sciencedaily.com/releases/2012/11/121122152928.htm Scientists have made a major advance in understanding how flu viruses replicate within infected cells. The researchers used cutting-edge molecular biology and electron-microscopy techniques to ?see? one of influenza?s essential protein complexes in unprecedented detail. The images generated in the study show flu virus proteins in the act of self-replication, highlighting the virus?s vulnerabilities that are sure to be of interest to drug developers.Thu, 22 Nov 2012 15:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121122152928.htmProtein folding: Look back on scientific advances made as result of 50-year old puzzlehttp://www.sciencedaily.com/releases/2012/11/121122152910.htm Fifty years after scientists first posed a question about protein folding, the search for answers has led to the creation of a full-fledged field of research that led to major advances in supercomputers, new materials and drug discovery, and shaped our understanding of the basic processes of life, including so-called "protein-folding diseases" such as Alzheimer's, Parkinson's and type II diabetes.Thu, 22 Nov 2012 15:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121122152910.htmStep forward in regenerating and repairing damaged nerve cellshttp://www.sciencedaily.com/releases/2012/11/121121145638.htm Researchers recently uncovered a nerve cell's internal clock, used during embryonic development. This breakthrough could lead to the development of new tools to repair and regenerate nerve cells following injuries to the central nervous system.Wed, 21 Nov 2012 14:56:56 ESThttp://www.sciencedaily.com/releases/2012/11/121121145638.htmArchitecture of rod sensory cilium disrupted by mutationhttp://www.sciencedaily.com/releases/2012/11/121121145621.htm Using a new technique called cryo-electron tomography, scientists have created a three-dimensional map that gives a better understanding of how the architecture of the rod sensory cilium (part of one type of photoreceptor in the eye) is changed by genetic mutation and how that affects its ability to transport proteins as part of the light-sensing process.Wed, 21 Nov 2012 14:56:56 ESThttp://www.sciencedaily.com/releases/2012/11/121121145621.htmAging: Scientists further unravel telomere biologyhttp://www.sciencedaily.com/releases/2012/11/121121130933.htm Researchers have resolved the structure of that allows a telomere-related protein, Cdc13, to form dimers in yeast. Mutations in this region of Cdc13 put the kibosh on the ability of telomerase and other proteins to maintain telomeres.Wed, 21 Nov 2012 13:09:09 ESThttp://www.sciencedaily.com/releases/2012/11/121121130933.htmDrug resistance biomarker could improve cancer treatmenthttp://www.sciencedaily.com/releases/2012/11/121121130811.htm Cancer therapies often have short-lived benefits due to the emergence of genetic mutations that cause drug resistance. A key gene that determines resistance to a range of cancer drugs has been reported in a new study. The study reveals a biomarker that can predict responses to cancer drugs and offers a strategy to treat drug-resistant tumors based on their genetic signature.Wed, 21 Nov 2012 13:08:08 ESThttp://www.sciencedaily.com/releases/2012/11/121121130811.htmGenome packaging: Key to breast cancer developementhttp://www.sciencedaily.com/releases/2012/11/121121130703.htm Two recent studies delve into the role of chromatin modifying enzymes and transcription factors in tumour cells. In one, it was found that the PARP1 enzyme activated by kinase CDK2 is necessary to induce the genes responsible for the proliferation of breast cancer cells in response to progesterone. In another, extensive work has been undertaken to identify those genes activated by the administration of progesterone in breast cancer, the sequences that can be recognized and how these genes are induced.Wed, 21 Nov 2012 13:07:07 ESThttp://www.sciencedaily.com/releases/2012/11/121121130703.htmShort DNA strands in genome may be key to understanding human cognition and diseaseshttp://www.sciencedaily.com/releases/2012/11/121121130643.htm Previously discarded, human-specific ?junk? DNA represents untapped resource in the study of diseases like Alzheimer?s and autism.Wed, 21 Nov 2012 13:06:06 ESThttp://www.sciencedaily.com/releases/2012/11/121121130643.htmBiomarking time: Methylome modifications offer new measure of our 'biological' agehttp://www.sciencedaily.com/releases/2012/11/121121130633.htm In a new study, researchers describe markers and a model that quantify how aging occurs at the level of genes and molecules, providing not just a more precise way to determine how old someone is, but also perhaps anticipate or treat ailments and diseases that come with the passage of time.Wed, 21 Nov 2012 13:06:06 ESThttp://www.sciencedaily.com/releases/2012/11/121121130633.htmKidney tumors have a mind of their ownhttp://www.sciencedaily.com/releases/2012/11/121121104552.htm New research has found there are several different ways that kidney tumors can achieve the same result -- namely, grow.Wed, 21 Nov 2012 10:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121121104552.htmMechanism to repair clumped proteins explainedhttp://www.sciencedaily.com/releases/2012/11/121121104416.htm Clumped proteins can be dissolved with the aid of cellular repair systems -- a process of critical importance for cell survival especially under conditions of stress. Researchers have now decrypted the fundamental mechanism for dissolving protein aggregates that involves specific molecular chaperones.Wed, 21 Nov 2012 10:44:44 ESThttp://www.sciencedaily.com/releases/2012/11/121121104416.htmNovel mechanism through which normal stromal cells become cancer-promoting stromal cells identifiedhttp://www.sciencedaily.com/releases/2012/11/121121104401.htm New understanding of molecular changes that convert harmless cells surrounding ovarian cancer cells into cells that promote tumor growth and metastasis provides potential new therapeutic targets for this deadly disease, according to new research.Wed, 21 Nov 2012 10:44:44 ESThttp://www.sciencedaily.com/releases/2012/11/121121104401.htmNew test for tuberculosis could improve treatment, prevent deaths in Southern Africahttp://www.sciencedaily.com/releases/2012/11/121120194932.htm A new rapid test for tuberculosis (TB) could substantially and cost-effectively reduce TB deaths and improve treatment in southern Africa -- a region where both HIV and tuberculosis are common.Tue, 20 Nov 2012 19:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121120194932.htmEvolution of human intellect: Human-specific regulation of neuronal geneshttp://www.sciencedaily.com/releases/2012/11/121120194926.htm A new study has identified hundreds of small regions of the genome that appear to be uniquely regulated in human neurons. These regulatory differences distinguish us from other primates, including monkeys and apes, and as neurons are at the core of our unique cognitive abilities, these features may ultimately hold the key to our intellectual prowess (and also to our potential vulnerability to a wide range of 'human-specific' diseases from autism to Alzheimer's).Tue, 20 Nov 2012 19:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121120194926.htmRibosome regulates viral protein synthesis, revealing potential therapeutic targethttp://www.sciencedaily.com/releases/2012/11/121120132906.htm Rather than target RNA viruses directly, aiming at the host cells they invade could hold promise, but any such strategy would have to be harmless to the host. Now, a surprising discovery made in ribosomes may point the way to fighting fatal viral infections such as rabies.Tue, 20 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121120132906.htmHow does antibiotic resistance spread? Scientists find answers in the nosehttp://www.sciencedaily.com/releases/2012/11/121120121835.htm Microbiologists studying bacterial colonization in mice have discovered how the very rapid and efficient spread of antibiotic resistance works in the respiratory pathogen, Streptococcus pneumoniae (also known as the pneumococcus). The team found that resistance stems from the transfer of DNA between bacterial strains in biofilms in the nasopharynx, the area just behind the nose.Tue, 20 Nov 2012 12:18:18 ESThttp://www.sciencedaily.com/releases/2012/11/121120121835.htmScientists identify inhibitor of myelin formation in central nervous systemhttp://www.sciencedaily.com/releases/2012/11/121120100155.htm Scientists have discovered another molecule that plays an important role in regulating myelin formation in the central nervous system. Myelin promotes the conduction of nerve cell impulses by forming a sheath around their projections, the so-called axons, at specific locations -- acting like the plastic insulation around a power cord.Tue, 20 Nov 2012 10:01:01 ESThttp://www.sciencedaily.com/releases/2012/11/121120100155.htm'Obese but happy gene' challenges the common perception of link between depression and obesityhttp://www.sciencedaily.com/releases/2012/11/121120084725.htm Researchers have discovered new genetic evidence about why some people are happier than others. The scientists have uncovered evidence that the gene FTO -- the major genetic contributor to obesity -- is associated with an eight per cent reduction in the risk of depression. In other words, it's not just an obesity gene but a "happy gene" as well.Tue, 20 Nov 2012 08:47:47 ESThttp://www.sciencedaily.com/releases/2012/11/121120084725.htmTelomere lengths predict life expectancy in the wild, research showshttp://www.sciencedaily.com/releases/2012/11/121119213144.htm Researchers have found that biological age and life expectancy can be predicted by measuring an individual's DNA. They studied the length of chromosome caps -- known as telomeres -- in a 320-strong wild population of Seychelles Warblers on a small isolated island.Mon, 19 Nov 2012 21:31:31 ESThttp://www.sciencedaily.com/releases/2012/11/121119213144.htmCancer: Some cells don't know when to stophttp://www.sciencedaily.com/releases/2012/11/121119171403.htm Certain mutated cells keep trying to replicate their DNA -- with disastrous results -- even after medications rob them of the raw materials to do so, according to new research.Mon, 19 Nov 2012 17:14:14 ESThttp://www.sciencedaily.com/releases/2012/11/121119171403.htmMultiple sclerosis ?immune exchange? between brain and blood is uncoveredhttp://www.sciencedaily.com/releases/2012/11/121119163301.htm DNA sequences obtained from a handful of patients with multiple sclerosis have revealed the existence of an ?immune exchange? that allows the disease-causing cells to move in and out of the brain.Mon, 19 Nov 2012 16:33:33 ESThttp://www.sciencedaily.com/releases/2012/11/121119163301.htmFruit fly studies guide investigators to molecular mechanism frequently misregulated in human cancershttp://www.sciencedaily.com/releases/2012/11/121119132056.htm Changes in how DNA interacts with histones ?- the proteins that package DNA ?- regulate many fundamental cell activities from stem cells maturing into a specific body cell type or blood cells becoming leukemic. These interactions are governed by a biochemical tug of war between repressors and activators, which chemically modify histones signaling them to clamp down tighter on DNA or move aside and allow a gene to be expressed.Mon, 19 Nov 2012 13:20:20 ESThttp://www.sciencedaily.com/releases/2012/11/121119132056.htm3-D light switch for the brain: Device may help treat Parkinson's, epilepsy; aid understanding of consciousnesshttp://www.sciencedaily.com/releases/2012/11/121119114249.htm A new tool for neuroscientists delivers a thousand pinpricks of light to individual neurons in the brain. The new 3-D "light switch", created by biologists and engineers, could one day be used as a neural prosthesis that could treat conditions such as Parkinson's and epilepsy by using gene therapy to turn individual brain cells on and off with light.Mon, 19 Nov 2012 11:42:42 ESThttp://www.sciencedaily.com/releases/2012/11/121119114249.htmNew factor of genetic susceptibility to Alzheimer's diseasehttp://www.sciencedaily.com/releases/2012/11/121119104944.htm A large-scale international study has just discovered a gene for susceptibility to a rare disease providing evidence of the heterogeneous aetiology of Alzheimer's disease.Mon, 19 Nov 2012 10:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121119104944.htmBlood cancer gene BCL6 identified as a key factor for differentiation of nerve cells of cerebral cortexhttp://www.sciencedaily.com/releases/2012/11/121119093848.htm The cerebral cortex is the most complex structure in our brain and the seat of consciousness, emotion, motor control and language. In order to fulfill these functions, it is composed of a diverse array of nerve cells, called cortical neurons, which are affected by many neurological and neuropsychiatric diseases. Researchers have opened new perspectives on brain development and stem cell neurobiology by discovering a gene called BCL6 as a key factor in the generation of cortical neurons during embryonic brain development.Mon, 19 Nov 2012 09:38:38 ESThttp://www.sciencedaily.com/releases/2012/11/121119093848.htmMinority report: Insight into subtle genomic differences among our own cellshttp://www.sciencedaily.com/releases/2012/11/121118141530.htm Scientists have demonstrated that induced pluripotent stem cells -- the embryonic-stem-cell look-alikes whose discovery a few years ago won this year's Nobel Prize in medicine -- are not as genetically unstable as was thought.Sun, 18 Nov 2012 14:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121118141530.htmSkin cells reveal DNA's genetic mosaichttp://www.sciencedaily.com/releases/2012/11/121118141524.htm The prevailing wisdom has been that every cell in the body contains identical DNA. However, a new study of stem cells derived from the skin has found that genetic variations are widespread in the body's tissues, a finding with profound implications for genetic screening.Sun, 18 Nov 2012 14:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121118141524.htmLikely basis of birth defect causing premature skull closure in infants identifiedhttp://www.sciencedaily.com/releases/2012/11/121118141432.htm Geneticists, pediatricians, surgeons and epidemiologists have identified two areas of the human genome associated with the most common form of non-syndromic craniosynostosis premature closure of the bony plates of the skull.Sun, 18 Nov 2012 14:14:14 ESThttp://www.sciencedaily.com/releases/2012/11/121118141432.htmDNA packaging discovery reveals principles by which CRC mutations may cause cancerhttp://www.sciencedaily.com/releases/2012/11/121117184658.htm A new discovery concerning a fundamental understanding about how DNA works will produce a "180-degree change in focus" for researchers who study how gene packaging regulates gene activity, including genes that cause cancer and other diseases.Sat, 17 Nov 2012 18:46:46 ESThttp://www.sciencedaily.com/releases/2012/11/121117184658.htmHepatitis C treatment's side effects can now be studied in the labhttp://www.sciencedaily.com/releases/2012/11/121116161059.htm Adverse side effects of certain hepatitis C medications can now be replicated in the lab, thanks to a research team. The new method aids understanding of recent failures of hepatitis C antiviral drugs in some patients, and could help to identify medications that eliminate adverse effects. The findings may aid the development of safer and more effective treatments for hepatitis C and other pathogens such as SARS and West Nile virus.Fri, 16 Nov 2012 16:10:10 ESThttp://www.sciencedaily.com/releases/2012/11/121116161059.htmReconsidering cancer's bad guyhttp://www.sciencedaily.com/releases/2012/11/121116124644.htm Researchers have found that a protein, known for causing cancer cells to spread around the body, is also one of the molecules that trigger repair processes in the brain.Fri, 16 Nov 2012 12:46:46 ESThttp://www.sciencedaily.com/releases/2012/11/121116124644.htmGene distinguishes early birds from night owls and helps predict time of deathhttp://www.sciencedaily.com/releases/2012/11/121116124551.htm New research shows that a gene is responsible for a person's tendency to be an early riser or night owl -- and helps determine the time of day a person is most likely to die.Fri, 16 Nov 2012 12:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121116124551.htmClues to cause of kids' brain tumorshttp://www.sciencedaily.com/releases/2012/11/121116091226.htm Insights from a genetic condition that causes brain cancer are helping scientists better understand the most common type of brain tumor in children.Fri, 16 Nov 2012 09:12:12 ESThttp://www.sciencedaily.com/releases/2012/11/121116091226.htmArthritis study reveals why gender bias is all in the geneshttp://www.sciencedaily.com/releases/2012/11/121115210541.htm Researchers have pieced together new genetic clues to the arthritis puzzle in a study that brings potential treatments closer to reality and could also provide insights into why more women than men succumb to the disabling condition.Thu, 15 Nov 2012 21:05:05 ESThttp://www.sciencedaily.com/releases/2012/11/121115210541.htmClass of RNA molecules protects germ cells from damagehttp://www.sciencedaily.com/releases/2012/11/121115172255.htm Passing one's genes on to the next generation is a mark of evolutionary success. So it makes sense that the body would work to ensure that the genes the next generation inherits are exact replicas of the originals. Biologists have now identified one way the body does exactly that.Thu, 15 Nov 2012 17:22:22 ESThttp://www.sciencedaily.com/releases/2012/11/121115172255.htmQuick test speeds search for Alzheimer's drugs: Compound restores motor function and longevity to fruit flieshttp://www.sciencedaily.com/releases/2012/11/121115152655.htm Researchers report that an efficient, high-volume technique for testing potential drug treatments for Alzheimer's disease uncovered an organic compound that restored motor function and longevity to fruit flies with the disease.Thu, 15 Nov 2012 15:26:26 ESThttp://www.sciencedaily.com/releases/2012/11/121115152655.htmProtein-making machinery can switch gears with a small structural change process; Implications for immunity and cancer therapyhttp://www.sciencedaily.com/releases/2012/11/121115133414.htm For the past several years, research has focused on the intricate actions of an ancient family of catalytic enzymes that play a key role in translation, the process of producing proteins. In a new study, scientists have shown that this enzyme can actually also work in another fundamental process in humans.Thu, 15 Nov 2012 13:34:34 ESThttp://www.sciencedaily.com/releases/2012/11/121115133414.htmPlant derivative, tanshinones, protects against sepsis, study suggestshttp://www.sciencedaily.com/releases/2012/11/121115133312.htm Researchers have discovered that tanshinones, which come from the plant Danshen and are highly valued in Chinese traditional medicine, protect against the life-threatening condition sepsis.Thu, 15 Nov 2012 13:33:33 ESThttp://www.sciencedaily.com/releases/2012/11/121115133312.htmStructure of enzyme topoisomerase II alpha unravelled providing basis for more accurate design of chemotherapeutic drugshttp://www.sciencedaily.com/releases/2012/11/121115132903.htm Medical researchers have for the first time described the structure of the active site core of topoisomerase II alpha, an important target for anti-cancer drugs. The type II topoisomerases are important enzymes that are involved in maintaining the structure of DNA and chromosome segregation during both replication and transcription of DNA. One of these enzymes, topoisomerase II alpha, is involved in the replication of DNA and cell proliferation, and is highly expressed in rapidly dividing cancer cells.Thu, 15 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121115132903.htmNewly discovered enzyme important in the spreading of cancerhttp://www.sciencedaily.com/releases/2012/11/121115132901.htm Enzyme hunters at UiO have discovered the function of an enzyme that is important in the spreading of cancer. Cancer researchers now hope to inhibit the enzyme.Thu, 15 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121115132901.htmGenetics point to serious pregnancy complication, pre-eclampsiahttp://www.sciencedaily.com/releases/2012/11/121115132613.htm New research has revealed a genetic link in pregnant moms - and their male partners - to pre-eclampsia, a life-threatening complication during pregnancy.Thu, 15 Nov 2012 13:26:26 ESThttp://www.sciencedaily.com/releases/2012/11/121115132613.htmMolecular mechanisms underlying stem cell reprogramming decodedhttp://www.sciencedaily.com/releases/2012/11/121115132344.htm Thanks to some careful detective work, scientist better understand just how iPS cells form ? and why the Yamanaka process is inefficient, an important step to work out for regenerative medicine. The findings uncover cellular impediments to iPS cell development that, if overcome, could dramatically improve the efficiency and speed of iPS cell generation.Thu, 15 Nov 2012 13:23:23 ESThttp://www.sciencedaily.com/releases/2012/11/121115132344.htmSurprising genetic link between kidney defects and neurodevelopmental disorders in kidshttp://www.sciencedaily.com/releases/2012/11/121115132342.htm About 10 percent of kids born with kidney defects have large alterations in their genomes known to be linked with neurodevelopmental delay and mental illness, a new study has shown.Thu, 15 Nov 2012 13:23:23 ESThttp://www.sciencedaily.com/releases/2012/11/121115132342.htmEven moderate drinking in pregnancy can affect a child's IQhttp://www.sciencedaily.com/releases/2012/11/121114172833.htm Relatively small levels of exposure to alcohol while in the womb can influence a child's IQ, according to a new study using data from over 4,000 mothers and their children.Wed, 14 Nov 2012 17:28:28 ESThttp://www.sciencedaily.com/releases/2012/11/121114172833.htmGene nearly triples risk of Alzheimer's, international research team findshttp://www.sciencedaily.com/releases/2012/11/121114171710.htm A gene so powerful it nearly triples the risk of Alzheimer's disease has been discovered by an international team of researchers. It is the most potent genetic risk factor for Alzheimer's identified in the past 20 years.Wed, 14 Nov 2012 17:17:17 ESThttp://www.sciencedaily.com/releases/2012/11/121114171710.htmDiscovery could lead to faster diagnosis for some chronic fatigue syndrome caseshttp://www.sciencedaily.com/releases/2012/11/121114171708.htm For the first time, researchers have landed on a potential diagnostic method to identify at least a subset of patients with chronic fatigue syndrome - testing for antibodies linked to latent Epstein-Barr virus reactivation.Wed, 14 Nov 2012 17:17:17 ESThttp://www.sciencedaily.com/releases/2012/11/121114171708.htm

Source: http://www.sciencedaily.com/rss/health_medicine/genes.xml

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Ashlee Simpson Spotted With Mystery Man Following Vincent Piazza Break-Up

Keeping somewhat of a low profile up until recently, singer Ashlee Simpson has returned to occupy the headlines once again with both the release of her new single ?Bat for a Heart? and her split from Boardwalk Empire?s Vincent Piazza. Simpson and Piazza have been dating since June 2011 but have now broken up over the Thanksgiving holiday, claiming that their busy schedules got in the way.? ?Vincent has been working a lot, so he hasn?t been able to spend that much time with Ashlee, which has caused some problems,? an undisclosed source informed Page Six of the New York Post.? Adding, ?It was a mutual agreement to split, but things have been bumpy for a little while, so perhaps they might reconcile.? There may be some truth to their decision to split.? At the moment the younger Simpson is gearing up to release her fourth studio album.? Could spending time in the studio and raising her son Bronx who she shares with Pete Wentz be to blame?? Or does it have something to do with the mystery man the singer was spotted with in New York.? Now it appears there is more to the story since Simpson was snapped [...]

Source: http://feedproxy.google.com/~r/RightCelebrity/~3/EIrSTT3kKq4/

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Thursday, November 29, 2012

Hurricane center pushes to improve storm surge warnings

MIAMI (Reuters) - Friday marks the end of an Atlantic-Caribbean hurricane season where the greatest devastation was caused by water rather than wind, U.S. National Hurricane Center Director Rick Knabb said.

Accordingly, the center is ramping up efforts to develop new warnings that better convey the threat from the deadly storm surge pushed ashore by monsters like Sandy, which slammed the U.S. Northeast in October.

"We've been working toward a new storm surge warning for a few years now," Knabb told Reuters in an interview at the hurricane center in Miami. "And Debby, Isaac and Sandy show us how much we really need to hit the accelerator on getting that storm surge warning out the door."

Debbie, Isaac and Sandy were three of the four storms that hit the United States during the 2012 season.

Starting with a meeting next week, the forecasters will review their warning systems and speed up development of a separate warning system for storm surge in hopes of having an experimental version ready to test in the next couple of years, Knabb said.

It will include a high-resolution graphic showing how high the surge would grow and how far inland it would reach at various times. Storm surge rarely correlates neatly with wind strength, Knabb said.

"Hurricane-force winds and storm surge doesn't always occur in the same places or at the same times when a storm approaches," he said. "Where the storm surge occurs is very dependent upon details of the coastline and the elevations and all that."

The United States is increasingly at risk from storm surge. Much of its densely populated Atlantic and Gulf coastlines lie less than 10 feet above sea level, and the seas are gradually rising as the Earth warms and ice caps and glaciers melt.

At the same time, the population in the hurricane region is growing rapidly. From 1990-2008, population density increased by 32 percent in Gulf coastal counties, 17 percent in Atlantic coastal counties, and 16 percent in Hawaii, according to the 2010 Census.

Much of the nation's commerce also depends on seaports and the transit systems that link to them.

Improving the storm surge warning system not only could help tailor evacuation orders as a storm approaches, it could help homeowners, business owners and governments know where and how to fortify before the next season comes.

"We can't hope that it doesn't happen again ... because it will. It's just a matter of when, not if," Knabb said.

Superstorm Sandy highlighted the need for more flexible warnings and greater focus on storm surge.

RAMPAGE

After killing 69 people on a rampage through the Caribbean, Sandy hit the densely populated northeastern United States in October, killing at least 131 more people and causing an estimated $71.3 billion of damage in New York and New Jersey.

It leveled entire beach towns, flooded subways and tunnels, paralyzed the nation's financial capital and knocked out power to 8.5 million customers in 21 states.

The behemoth storm was more than 900 miles wide as it churned northward up the Atlantic. It approached New Jersey as a hurricane, a spiraling system that draws energy from warm seas and has its strongest winds around the center.

Just offshore, it morphed into an extra-tropical "Nor'easter," a colder storm that draws energy from the atmosphere and spreads the strongest winds over a broader area. That did not lessen the danger.

"The ocean was churned up over a long period of time by this massive wind field. Tropical or not, we knew the storm surge hazard was going to be significant," said Knabb, who had warned for days that Sandy would push a surge of seawater up to 11 feet high over parts of New York and New Jersey.

"That's what did most of the damage in Sandy," he said. "We had a huge area impacted by storm surge and waves and most of that area did not get hurricane-force winds."

The hurricane center worked closely with local government officials who ordered evacuations in low-lying areas, and waged an intense media campaign to get the warnings out.

But since Sandy was not expected to be a hurricane or tropical storm at landfall, it didn't fit neatly into the hurricane center's standard package of warnings and graphics that focus on wind.

Hurricane watches and warnings, which are issued by local governments on the advice of forecasters, are based on expectations for strong winds within 36 to 48 hours.

The forecasters did not want hurricane warnings issued only to have them taken down when Sandy made its anticipated transition to an extra-tropical storm near the shore. They feared that would create a false impression that the danger was over just when it was greatest.

"It would have really caused a lot of confusion for a lot of people trying to make decisions and evacuate," Knabb said.

Another option - "faking it" and pretending it was still a hurricane when it wasn't - would have been an obvious deception, Knabb said.

"We do need to have some credibility and call it like we see it," he said.

Instead, the hurricane center continued to issue advisories in the regular format and through its regular distribution system, stressing the storm surge danger and referring people to their local Weather Service office for specific flood projections.

In all, the 2012 season brought 19 tropical storms, 10 of which strengthened into hurricanes.

That made it a busier-than-average year that topped the seasonal forecasts issued in the spring by government and private meteorologists.

But none of the storms that hit the United States this year were "major" hurricanes on the Saffir-Simpson scale that ranks them into five categories according to their wind strength. Most were not even hurricanes, which are tropical cyclones with sustained winds of at least 74 miles per hour.

Beryl was a tropical storm when it swirled ashore in Florida and soaked the southeastern United States in May, before the season officially began. Debby was also a tropical storm when it dumped 2 feet of rain over northern Florida in June, sending thousands fleeing from rapidly rising rivers.

Tropical Storm Isaac delayed the start of the Republican National Convention when it brushed by Tampa, Florida. It strengthened into a hurricane that flooded the Mississippi and Louisiana coasts and put New Orleans' refurbished levees to the test.

Despite storm surges that topped 12 feet, the elaborate defenses built to protect the city after the 2005 Hurricane Katrina disaster withstood the onslaught.

(Reporting by Jane Sutton; Editing by Vicki Allen)

Source: http://news.yahoo.com/hurricane-center-pushes-improve-storm-surge-warnings-174150385.html

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Argentina wins debt reprieve, default averted for now

NEW YORK (Reuters) - Argentina has won a reprieve against having to pay $1.33 billion next month to "holdout" investors who rejected a restructuring of its defaulted debt and have waged a long legal battle to be paid in full.

A U.S. appeals court granted an emergency stay order on Wednesday that gives Argentina more time to fight a debt ruling favoring the holdout creditors and eases investor fears of a new default as early as next month.

Last week, U.S. District Judge Thomas Griesa ordered Argentina to deposit the $1.33 billion payment by December 15 for investors who rejected two restructurings of bonds left over from its massive 2002 default.

Griesa's order raised the risk of a technical default on about $24 billion worth of debt because it meant that if Argentina sticks to its position of not paying the holdouts, it would also be barred from paying investors who agreed to take a severe haircut in two debt exchanges in 2005 and 2010.

Branding the holdouts as "vulture funds," the government has vowed never to pay them and it swiftly appealed Griesa's ruling.

Argentina argued that, if left to stand, the order would make future restructurings impossible for countries facing debt crises because creditors would have no incentive to exchange their bonds at a discount.

However, some legal experts said Griesa's order would not have such broad ramifications because Argentina hurt its own cause in refusing to pay the holdouts, and that Griesa's ruling focused on the government's behavior in this specific case.

Griesa has often voiced frustration with Argentina in court and was riled by recent statements from government officials saying that the country would defy his orders.

In its decision on Wednesday, the 2nd U.S. Circuit Court of Appeals put off until well into 2013 a ruling on whether or not Argentina will have to pay in full the holdouts who refused to participate in its two restructurings, which paid less than 30 cents on the dollar.

"The extension of the stay brings back rationality and due process to a litigation that was being rushed through in a manner that understated the importance of the huge precedent that the district judge was seeking to set," said Vladimir Werning, an emerging markets economist at JPMorgan in New York.

Both Argentina and bondholders who took part in the debt exchanges filed appeals to the 2nd Circuit against Griesa's order. The appeals court will hear oral arguments on February 27.

Argentina's economy ministry said the appeal court's decision "ensures normal performance of Argentina's debt payments in December."

"The threat of default has been removed for now," said Ignacio Labaqui of emerging markets consultancy Medley Global Advisors.

"This is really good news for Argentina and exchange bond holders," he added. "The ruling came faster than expected, which sends the message that Griesa's decision may have been too harsh, from the point of view of the appeals court."

Lead holdout investors Elliott Management Corp and Aurelius Capital Management both declined to comment.

Lawyers for the holders of Argentina's exchanged bonds, who will now be paid a combined $3.3 billion in December as scheduled, welcomed the 2nd Circuit's order.

"The stay ensures that the exchange bondholders will receive their rightful payments through December, and until the Court can carefully consider the significant issues and interests that are involved before rendering its final ruling," said the statement from law firm Boies Schiller & Flexner LLP.

(Additional reporting by Basil Katz and Nate Raymond in New York and Hugh Bronstein in Buenos Aires; Editing by Kieran Murray)

Source: http://news.yahoo.com/appeals-court-gives-argentina-reprieve-debt-case-005328259--sector.html

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iOS 7 wants: Better email attachment handling

iOS 7 wants: Better email attachment handling

Almost 6 years after it was introduced, you still can't attach files to emails in iOS. While iOS 6 has introduced a method to attach photos and videos to in-progress emails, it suffers from poor discoverability, and only works with content from the Photos app. If you want to attach any other file to an email, it's a usability disaster.

Adding photo or video attachments to in-progress emails is done via the same pop-up menu originally introduced in IOS 3 for cut, copy, and paste. You have to tap the screen to get the popup, tap a tiny, obscure arrow button to get more options, and then tap to add the attachment.

An easy to find, easy to use attachment button would be simpler. It's a solution employed by third-party apps like Facebook, email clients like Gmail and Sparrow, and the official Twitter app and Tweetbot.

Some of these are more elegant than others, but all of them are more discoverable. And that's still only for photos and videos. What about files?

Emailing, and including files as attachments in email, is a common task and something that takes only a few seconds on the Mac with OS X. Trying to attach a non-photo or video file on an iPhone or iPad with iOS takes an annoying amount of time and causes an unreasonable amount of frustration. Here's some blog-theater by way of example:

"Hey, Rene, can you email me the dates for that trip?"

"Sure, Kevin." I grab my iPhone, open the Mail app, add Kevin as the recipient, add the subject "trip", paste in the dates, then--

"Could you also attach that outline for discussion topics?"

"Um..."

My only option now is to copy the contents of the email, trash it, go to the app I wrote the topics in, find the file, tap share, tap email, add Kevin again, add the subject again, paste in the dates again--

Shit. I deleted the app I wrote the topics in. A hotter, newer app came out and I started using that instead, and even though both use iCloud, neither has any idea the other exists so... I re-download the old app and pray the data is either still there, or magically comes back from the cloud.

"And those two PDF files about that thing?"

Double shit. Both those PDF files are in different PDF apps, one in a simple reader, the other one in an app that supports annotation. Now I have to send the discussion topics from one app, and each of the PDF files from their apps. That's three separate emails, and nothing approaching a thread.

Now Kevin is laughing his ass off at me and asking me to tell him again how the iPhone is easy to use, and I want to punch things.

And the reason for all this is that Apple forgot a cardinal principle of design: unreasonable simplicity sometimes leads to inane levels of complexity.

Apple already uses a blue + button to add contacts. Something like that could work for attachments as well. (I did a mockup along those lines for a pre-iOS 6 article on higher hanging fruit, but I'll update it below.)Tapping the attachment button could bring up a Share Sheet that includes the Photos icon, so you could attach photos as normal, but also any other app icon that contains documents. Tapping an icon would bring up a list view of all the files associated with that app. It's a bit overwrought, but it fits with the existing iOS constructs, including the binding of files to apps, and has the advantage of familiarity-as-a-feature.

Depending on how many file-capable apps are installed, it could create a very dense Share Sheet. It also still requires the user remember which app contains which file.

That's why, for the last couple of years, I've been asking for a simple, flat document repository for iOS -- a Files app that works the same way as the Photos app (or Passbook app for that matter). A Files app for iOS would remove unnecessary cognitive load from users and solve a wide swathe of current usability problems with iOS, including email attachments.

With a document repository, any user file could be attached to any in-progress email, without the need for a Share Sheet, or for the user to remember app ownership. And it would do so in a way that's consistent with how iOS already works, increasing simplicity at the same time.



Source: http://feedproxy.google.com/~r/TheIphoneBlog/~3/uw9GcPdDGvs/story01.htm

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Video: Which guest TODAY weatherman read it best?

Sorry, Readability was unable to parse this page for content.

Source: http://video.today.msnbc.msn.com/today/49978779/

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Wednesday, November 28, 2012

Nintendo's Wii U Starts Strong - NYTimes.com

In its first week on the market, Nintendo?s new videogame console, the Wii U, sold about as well in the United States as the original Wii did at the same point in its life, according to Nintendo.

In an interview, Reggie Fils-Aime, president of Nintendo of America, estimated that the company sold more than 400,000 Wii U?s here from the product?s introduction on Nov. 18 and last Saturday. Mr. Fils-Aime said that figure was based on sales data Nintendo collected electronically from retailers. He said total American sales for the product were likely higher.

A more complete sales figure for Wii U will come soon when NPD, a research firm that estimates retail sales, releases data for the product?s first week on the market, Mr. Fils-Aime said. He said 475,000 of the original Wiis were sold during the product?s first week on the market in 2006, citing NPD data.

The results are a good sign for the company?s latest product. The Wii was an enormous hit that outsold competing systems from Microsoft and Sony, but its sales have faded in recent years, putting pressure on the Wii U to help lead a turnaround at Nintendo.

?We are essentially sold out at retail,? Mr. Fils-Aime said. ?As soon as we replenish product to retailers, it sells out immediately.?

It?s important to keep in mind that supplies of new game consoles are often very limited when they first go on sale, as manufacturers ramp up production. Nintendo has a very dedicated audience that craves almost anything new the company has to offer, not unlike Apple?s fans. The real test of the Wii U?s durability will come when the product is in better supply and more casual gamers, who don?t dream about Mario and Zelda in their sleep, can more easily buy it.

Mr. Fils-Aime said another of Nintendo?s more recent products, the handheld Nintendo 3DS, was also selling well, racking up sales of 6.4 million units in the United States during its first 21 months on the market. He said Nintendo had sold one million more Nintendo 3DSes than it did an older handheld, the Nintendo DS, during the comparable time period.

Source: http://bits.blogs.nytimes.com/2012/11/26/nintendos-wii-u-starts-strong/

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World's first toilet park flush with success in Korea

SUWON, South Korea (Reuters) - Rodin's Thinker is pondering even harder than usual as he sits astride a toilet at what has been dubbed the world's first theme park dedicated to the humble restroom - a monument to one South Korean man's vision.

The park, located about an hour outside of Seoul in the city of Suwon - otherwise known as the home of Samsung Electronics - centers around a toilet-shaped museum building that was once the home of Sim Jae-duck, founder and first president of the World Toilet Association.

Legend has it that Sim, a former Suwon mayor who made his fortune with a metal products business and was dubbed "Mr Toilet," was born in his impoverished grandmother's outhouse.

"He is a man whose life literally began in a toilet and ended at a commode-shaped house," said Lee Yeun-sook, manager of planning at the "Mr Toilet Sim Jae-duck Foundation".

Sim, who died in 2009 at the age of 70, shot to fame in South Korea when he provided loos for soccer fans when the country hosted the 2002 World Cup.

The organization he founded has as its mission spreading the benefits of hygienic toilets around the world, joining the like-minded World Toilet Organization based in Singapore.

Before Mr.Toilet's house was donated to Suwon city, visitors could book it for an overnight stay, but at the cost of $50,000 a night - the charge to raise money for a toilet building charity. There were no takers.

Other exhibits at the park include Korean traditional squat toilets, European bedpans, and Marcel Duchamp's sculpture "Fountain," a porcelain urinal.

Suwon has since dubbed itself the mecca of toilet culture and has pushed to get toilets recognized as a central part of everyday life. It has funded toilet building programs in developing countries such as the Philippines.

At home, toilet conditions have rapidly improved as South Korean living standards shot from poverty to riches in a generation.

"For our generation, a toilet was a very dirty and smelly place where you never wanted to go," said Kim Gye-soon, a 52-year-old tourist at the theme park. "But now it is totally different."

Suwon will continue the life-work of one of its most famous sons by constructing a toilet culture center in 2014 near the current park, which has attracted about 40,000 visitors since it opened in July.

Like many of the best things in life, the toilet museum is free.

"Going to the restroom is as vital as eating. In a sense, nations and governments should work to make sure everyone has an equal access to toilets and feels happiness in there," said Lee.

(Reporting By Jane Chung, editing by Elaine Lies)

Source: http://news.yahoo.com/worlds-first-toilet-park-flush-success-korea-032837059--finance.html

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Tuesday, November 27, 2012

Was Mom nuts? Scary movies we saw as kids

Paramount Pictures

Scarlett Johansson saw "Psycho" way too early. She's not the only adult who remains scarred by scary movies seen as kids.

Scarlett Johansson is starring in the new movie "Hitchcock." and confessed on TODAY that she watched the great director's "Psycho" at a far-too-early age -- 7.

"I was raised on Hitchcock films," she said on the show. "My mother is a huge film buff and I saw 'Psycho' probably when I was too young, I mean I was like 7 or 8 and it was incredibly traumatizing. ... It petrified me."

Turns out Johansson is far from alone. Members of our staff were quick to share their own experiences with too-early scares. Seems that you never forget the title of your first cinematic chill.

Not every movie was meant to scare the youngster in question. "I remember my dad showing me 'Swamp Thing,'" confessed TODAY Moms editor Rebecca Dube. "He thought it was funny but my 8-year-old self did not appreciate the camp hilarity and found it to be PURE HORROR. Nightmares for months...."

Warner Bros.

"Mine was 'The Deep' when I was 9 and OMG HE IS DRAWING ON HER STOMACH IN BLOOD WITH A CHICKEN FOOT but I?m sure it didn?t affect me at all," said Gael Fashingbauer Cooper, movies editor.

"My best friend saw the scary movie ?It's Alive? when we were around 10 and wouldn?t sleep in her room alone for 6 months," shared senior multimedia editor Mish Whalen.

Some inappropriate films weren't horror, but were still too adult for the child in question. "I saw 'Raging Bull' pretty young," said entertainment editor Kurt Schlosser. "Take that for what it?s (expletive) worth."

Did you see a scary or otherwise too-adult movie when you were way too young for it? Share the title, and the circumstances (Nightmares? Did Dad get in trouble with Mom for taking you?). Tell us on Facebook.

Related content:

Source: http://todayentertainment.today.com/_news/2012/11/27/15486870-were-our-parents-nuts-johansson-not-only-one-scarred-by-inappropriate-movies-as-a-kid?lite

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Now Obama Wants Your 401(K)

Barack Obama between flags SC Now Obama Wants Your 401(K)

NEW YORK ? Two years ago, as WND reported, the Obama administration was proceeding with a novel way to finance trillion-dollar budget deficits by forcing IRA and 401(k) holders to buy Treasury bonds by mandating the placement of government-structured annuities in their retirement accounts.

Remarkably, those financial professionals specializing in private retirement savings and the U.S. citizens investing in private retirement plans now face the possibility the Obama administration and its allies on the political left will impose rules and regulations that effectively abolish the private retirement savings and investment markets.

Recent evidence suggests government officials continue to eye the multi-trillion dollar private retirement savings market, including IRAs and 401(k) plans, eyeing the opportunity to redistribute private retirement savings to less affluent Americans and to force the retirement savings out of the private market and into government-controlled programs investing in government-issued debt.

Government takeover?

An Investment Company Institute study published this month found that U.S. retirement assets totaled $18.5 trillion at the end of the second quarter 2012, of which 3.5 trillion was in IRAs and $5.1 trillion was in 401(k) plans.

Since 2010, the U.S. Treasury Department and the Department of Labor have been holding combined hearings on various plans designed to introduce government-mandated retirement plans and investment options, including government annuities invested primarily in U.S. Treasury debt, into the private retirement savings market.

Read More at WND?.?By Jerome R. Corsi.


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Source: http://www.westernjournalism.com/now-obama-wants-your-401k/

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GLHFCasting ? Home Improvement Professionals

Home Improvement Clayton Are you in the shadow of brand-new homes that have your place looking decades behind? Now is the time to start thinking about a home improvement project. The beauty of improving your home is that you can begin almost anywhere. You can choose the project that is the greatest need or what can keep you under budget easily. These projects could include fixing a deck, remodeling a bathroom or kitchen, or giving the living room new paint or wallpaper. Professional home improvement specialists can guide you to find the right materials and show you the best way to get the job done right. Professional Home Repair

Source: http://www.glhfcasting.com/home-improvement-professionals-4/

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Nexus 7 32GB hits '1 Sale A Day' for $249.99 with free shipping

Nexus 7

It's that time of year where we start seeing some great deals on smartphones and tablets. We saw the Nexus 4 go up for a "discounted" (if you don't count the Play Store price) $499 yesterday, but we have a deal here regardless of what you compare it to. The Nexus 7 has been around long enough that we're starting to see it pretty consistently discounted on various sites, but the savings here come from your ability to save on shipping (and often sales tax as well) over what Google Play charges. Might just save you 10- or 15-percent overall.

Take a look at the source link below if you're interested in grabbing one of these at a great price.

Source: 1 Sale A Day



Source: http://feedproxy.google.com/~r/androidcentral/~3/m_YGnZTHXLg/story01.htm

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Worried For Decreasing Top International Cbse School Admissions ...

In the today's highly competitive scenario, getting your child admitted to best schools has become one of the most challenging tasks for parents. The mad rush for school admissions at the Top School In India has already started. The majority of parents seems to be busy these day in order to secure an admission in the best schools in India. With the fast approaching next academic sessions some children got admissions whereas others will have to knock on the doors of several schools for a seat. Above all, the shrinking school admissions options has emerged as a another big problem with parents


If you're amongst those fractions of parents who failed to register their kid's in the top Cbse school Bangalore and quite worried about the shrinking school admissions options then Global Indian International School can bring you some relief. Shares close proximity to Whitefield, GIIS Bangalore is one of the most prestigious Top International School Bangalore invites application for admission to nursery to class 5th.? To minimize the hurdles of school admissions, GIIS Whitefield Bangalore has started Online School Admissions so that parents can apply for admissions hassle-free with an ease of the internet. An initiative of online school admissions standout as a quick and time-saving option.

Well acknowledged as the axiom in the school education, GIIS Bangalore is counted amongst the Best Bangalore Cbse Schools promises the finest quality of school education and nursery education to your child. Many parents consider GIIS Bangalore as the Top Cbse International School Bangalore offering Central Board of Secondary Education (CBSE; India's leading curriculum), and the Global Montessori Plus Programme.? It is an initiative of Global Schools Foundation - the Singapore-based school education chain was founded in 2002, established with a focused dedication towards shaping well-rounded global citizens.

Key Highlights of Global Indian International School Whitefield Bangalore:

1.?????? Work under the guiding principals of the 'Global Centre for Education Excellence' (GCEE) based on the Malcolm Baldrige National Quality Programme of USA. The GCEE model greatly focuses to serve as a working tool for understanding and managing performance and guide proper planning of opportunities for learning.

2.?????? Have the Mahatma Gandhi Centre for Values and Thoughts (MGCVT) that gives GIIS students valuable insights into the experiences and teachings of Mahatma Gandhi.

3.?????? The school adopts a futuristic curriculum to impart 21st century skills, so that students can emerge as confident, competent and contributory global citizens.

4.?????? It strongly focus on integrating formal with informal learning, helps learners acquire the necessary skills for inter-disciplinary thinking, critical thinking, problem solving and resolving conflict.

5.?????? It's multi-faceted learning methods address the physical, mental, emotional, psychic, intellectual and spiritual domains of the individual learner, thus acknowledging the uniqueness of each.

6.?????? It comes with power-packed curriculum warrants meaningful instruments for learning that would provide the necessary intellectual, emotional, psychological and spiritual power to the learner that makes him/her a confident, competent and contributive citizen of the society.

Admissions are going in the Top Cbse International School Bangalore - GIIS Bangalore for Pre Nursery, Nursery to Class 5th. Admission process of Global Indian International School is very simple. The GIIS admission criteria are as follows :

?????????????? 2.5 years complete for Nursery on 31stMay,2013

?????????????? 3+ years complete for Nursery on 31st May, 2013

?????????????? 4+ years complete for KG on 31st May,2013

?????????????? 5+ years complete as for Std 1? on 31st May, 2013

Registration forms for school admissions to top Bangalore schools are available online. All? interested parents can also apply for admissions to classes Nursery to V online through online application form (only if your child fulfilling admission eligibility conditions). Parents need to submit online GIIS Enquiry Form and then, guardians are expected to fill the admission form and submit all the important documents (such as passport size photo, medical reports, birth certificate, etc.) at the time of admission.

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Article Tags : top Cbse school Bangalore, Top School In India, Bangalore Cbse Schools

Source: http://www.workoninternet.com/business/reviews/computers-internet/education/220192-worried-for-decreasing-top-international-cbse-school-admissions-options.html

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Monday, November 26, 2012

Gearbox's Randy Pitchford on Aliens and the future of game consoles

Digital Trends sits down with Randy Pitchford to talk about developing a proprietary engine to bring the world of Aliens: Colonial Marines to life. Plus: Gearbox's president on the future of video game consoles.

Randy Pitchford had a good year. In the thirteen years since founding his studio, Gearbox Software has never had a hit quite like Borderlands 2, one of the best selling and widely praised video games to come out in a very crowded year. What?s more his studio?s long labor of love, Aliens: Colonial Marines, is just three months away from release after a nearly six year development. Where some game makers are itching for the next console generation to start so it can ramp up the declining console market, Pitchford and his team of creators are sitting pretty.

Digital Trends caught up with Pitchford at a recent event to talk about the prolonged development of Aliens, the unique technology powering that game, and the future of how we play video games.

Aliens: Colonial Marines uses a new engine. Why? Why not use something simple like Unreal Engine 3 to speed up development?

We haven?t really talked about it much, but we made a proprietary lighting engine for this game. Even though we?re at the end of this console generation, because this is a deferred rendering engine it?s going to give us a good sense of what next gen games are going. Deferred rendering is the future. We do leverage Unreal for the architecture and the data sets, for a lot of the under the hood stuff, but in terms of graphics, that?s the stuff we created just for this.

The Aliens films, to do that authentically, we needed deferred rendering. We needed dynamic lights and shadows. In the current generation, for games without a non-deferred renderer, all of the lighting is sort of baked into the environment which means it?s static. Things can?t move. Now take the scene in the Sulaco: There are so many things shifting around, with crates sliding across the room, ship rocking and lolling. If things move, the lights all need to work. When stuff?s blowing up, when you?re on the surface of LV-426, the atmosphere is alive. We needed that to feel natural, so we needed the lighting to be rendered in real time with the game. If you think about some of the other stuff we?re doing like Borderlands, it has a totally different look and feel. Our Brothers in Arms franchise, it?s a totally different environment and feel. Aliens has a very unique art direction, so it demanded custom technology.

You said the team met with Ridley Scott to discuss doing the game before it went into production. When did that meeting happen?

I think that was May of 2006. Maybe 2005.

At what point in the past seven years then did you decide to build this unique technology?

The meeting was 2005 or 2006, but the deal didn?t happen for another year or so after that.

gearbox software presidentThat?s when Sega announced it had the Alien franchise license.

We hadn?t even written the first line of code and Sega was so excited that they and Fox had to turn around and put out a press release! (laughs) It was a business statement, but if you?re an Aliens fan like me and you hear, ?There?s going to be a sequel to the movies as a video game and Gearbox is making it!?? suddenly there?s this expectation. I want that now! Usually when we make a game, we?ll be all the way through pre-production and into production before an announcement is made.

It was after our prototype, after our pre-production, that John Cavanagh, one of our programmers, took on the task of building the new renderer for this game. I believe we?ll be using this technology for a while. It?s going to be very useful in the future.

So that was the end of 2008 when we started making that technology and it was probably finished by the end of 2010.

Speaking of 2008, that was when you announced that Battlestar Galactica writers David Weddle and Bradley Thompson would be working on Aliens: Colonial Marines. What role did they play?

Those guys are awesome! We had a framework and sketches of characters when we started the game. Those guys came in and wrote all of this material that brought the characters to life for us. So now we don?t have to think about what we want the characters to be, we think, ?Okay, in this situation, how would Bella react to this? What would Winter say?? We know the answer because those guys helped us flesh everything out. Amazing, amazing work. They wrote enough material that we could have an entire series of television, honestly. I loved working with them. [Gearbox writer] Mikey Neumann was able to work very closely with them, and he was able to adapt what they did and write it to a video game, which is very different than writing for a passive movie. The quality that we?ve gotten in the narrative was the result of that iterative process and David and Bradley were instrumental in that. It was so cool to work with them.

I?m not sure if you?re office is still like this, but I saw a picture of it once and the game console collection on display in there is impressive to say the least. You clearly love this old, devoted technology. Aliens, Borderlands, all of Gearbox?s games really are clearly built as multiplatform games, both PC and console. My question for you: What?s the future of the console? Do we need them anymore?

How far in the future are we talking about?

Say fifteen years.

Obviously power is great. There are two components to this. One is energy drawn from whatever source you get it from. In fifteen years, wireless power still won?t be ubiquitous. We?ll be experimenting with it in fifteen years and some people will probably have appliances in their garages that allow their cars to charge wirelessly, but we won?t have ubiquitous wireless power for all devices. So in fifteen years, we?ll still see an advantage in things that can plug into a wall, a devoted power source, and draw a tremendous amount of energy.

The more energy we can draw, the more fuel we have for computing power. Computing power takes energy. Computing power will always allow us to simulate things, whether it?s visuals or gameplay or logic or physics. The more computing power we have, the more robust that simulation will be. Within fifteen years, we?ll still be using our senses?touch, sight, sound?to consume that simulation. So the processing power we?ll be using will still be simulating something that reaches us through our normal senses. We won?t be at a point where our senses can be tricked into something that?s like the holodeck or when something?s plugged right into our brain, where there?s no interpretive layer between the simulation and perception, that?s not going to happen yet.

Within that construct, power will still be a factor and power will let us do all kinds of things. We?re going to see some new fronts in the next fifteen years that are kind of difficult to imagine when you just think, ?Okay, I?ve got a dedicated machine, I?ve got a portable thing that uses a battery like a phone or handheld, I?ve got large format stuff like in a movie theater.? You?re going to see that whole spectrum, and it will largely be the same. It will be an iteration of how we understand entertainment, but it?ll be a massive iteration. You?ll see between twelve and twenty-four times the complexity possible on today?s machines.

Today, though, what does having your game on a console give you? What?s the benefit of the technology?

One of the advantages that consoles have for a customer is that you can trust it. It?s a simple price, an entry point that?s accessible, and you can plug it in and it all works. It rides this balance where one step up is a PC that draws a lot more power and a big step up in visuals, but a lot more complexity since it?s a moving platform. That makes it not so comfortable. The dedicated platform is fixed, though, which lets us developers make tremendous assumptions about what the customer is using. Because of those assumptions, we can commit ourselves, we can be very efficient in what we make.

Consoles let you make those assumptions. Look at the iPhone. Because there are now several generations of iPhone, in creating software we can either choose to eliminate a percentage of the potential user base by making our game only compatible with the iPhone 4 or 5 or we can hold ourselves back and cover the whole spectrum. Even that fragmentation changes our efficiency and quality from what it would be if we had a single, unchanging platform.

Because the phone cycle is ramping up so quickly, our only choices are abandon our software, iterrating it, or making new software. With a long console cycle, we can really commit ourselves to that single fixed platform and be super optimal. You can feel it in the results. We?re doing things on these platforms that are a lot more incredible than what we saw at the beginning of the generation.

It really doesn?t matter, though. At the end of the day in the entertainment business, what you care about is how many people can I reach and to what extent can I satisfy them. Our mission is to make people happy. We want to entertain people wherever they are.

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Source: http://www.digitaltrends.com/gaming/gearbox-software-president-randy-pitchford-on-aliens-colonial-marines-and-the-future-of-game-consoles/

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